DISEASE: Bacterial dieback of nectarine
HOST: Nectarine
Discolored tissues of crown and roots were caused by systemic invasion of the pathogen.
Bacterial dieback of nectarine | Nectarine
DISEASE: Bacterial dieback of nectarine
HOST: Nectarine (Prunus persica 'Fantasia')
PATHOGEN: Pseudomonas syringae pv. persicae
SOURCE: J. Young
DISEASE: Oleander knot
HOST: Oleander
Oleander with small knots/galls on leaf and stems.
Oleander knot | Oleander
DISEASE: Oleander knot
HOST: Oleander (Nerium oleander)
PATHOGEN: Pseudomonas savastanoi pv. nerii
SOURCE: W. Sinclair
DISEASE: Oleander knot
HOST: Oleander
Young stem with knots/galls.
Oleander knot | Oleander
DISEASE: Oleander knot
HOST: Oleander (Nerium oleander)
PATHOGEN: Pseudomonas savastanoi pv. nerii
SOURCE: R. Raabe
DISEASE: Oleander knot
HOST: Oleander
Leaf with early stage of knot/gall formation.
Oleander knot | Oleander
DISEASE: Oleander knot
HOST: Oleander (Nerium oleander)
PATHOGEN: Pseudomonas savastanoi pv. nerii
SOURCE: R. Raabe
DISEASE: Oleander knot
HOST: Oleander
Infected flowers with small knots/galls and necroses.
Oleander knot | Oleander
DISEASE: Oleander knot
HOST: Oleander (Nerium oleander)
PATHOGEN: Pseudomonas savastanoi pv. nerii
SOURCE: W. Sinclair
DISEASE: Oleander leaf scorch
HOST: Oleander
Diseased plant with scalloped, yellowish leaves. This usually is followed by dieback of twigs and branches. Plants often die 2 years after infection.
Oleander leaf scorch | Oleander
DISEASE: Oleander leaf scorch
HOST: Oleander (Nerium oleander)
PATHOGEN: Xylella fastidiosa
SOURCE: S. Purcell
DISEASE: Oleander leaf scorch
HOST: Oleander
Oleander with symptoms of severe scorch.
Oleander leaf scorch | Oleander
DISEASE: Oleander leaf scorch
HOST: Oleander (Nerium oleander)
PATHOGEN: Xylella fastidiosa
SOURCE: M. Blua
DISEASE: Oleander leaf scorch
HOST: Oleander
Oleander with scorch symptoms primarily at leaf tips, an early stage of disease.
Oleander leaf scorch | Oleander
DISEASE: Oleander leaf scorch
HOST: Oleander (Nerium oleander)
PATHOGEN: Xylella fastidiosa
SOURCE: M. Blua